جنسی جذبہ

پیارے دوستو! اسلام علیکم

ہر انسان کے اندر خدا نے ایک طاقت پیدا کی ہے جس کو جنسی جذبہ کا نام دیا جاتا ہے۔ اس طاقت کے متعلق میرے پاس جو واضح نظریہ موجود ہے وہ صرف ابھی تک یہی ہے کہ اس کے ذریعے انسان کی نسل چلتی ہے۔
لیکن میں نے اس طاقت یا جذبہ کے متعلق کچھ اور بھی پڑھا ہے۔لیکن میں ان باتوں کو سمجھ نہیں سکا۔ سوال یہ ہے کہ یہ تو بہت واضح ہے کہ اس طاقت یا جذبہ کے ذریعے انسان اپنی نسل کو آگے چلاتا ہے لیکن یہ کیسے ممکن ہوتا ہے کہ ایک انسان کے اندر
اس طاقت یا جذبہ کے ذریعے جسمانی طاقت پیدا ہوتی ہے۔یہ جنسی طاقت کیسے پیدا ہوتی ہے؟
دوسری بات یہ کہ
یہ کیسے ایک بندے کے اندر ذہنی ارتکاز۔ یکسوئی پیدا کرتی ہے۔اس جذبہ کے ذریعے ذہنی ارتکاز کیسےممکن ہوتا ہے؟
تیسری بات یہ کہ جس کا آپ نے جواب ڈھونڈنا ہے کہ
یہ کیسے ممکن ہے کہ ایک بندہ اپنے جنسی جذبہ کو کم از کم دو ماہ تک کنٹرول کر سکے؟
الحمداللہ میں ایک مسلمان ہوں۔ ہو سکتا ہے کہ کئی دوست مجھے سب سے پہلا مشورہ یہ دیں کہ میں روزے رکھوں۔ اس سلسلےمیں میں یہ بتا دوں کے روزے کے ذریعے جنسی جذبہ کنٹرول نہیں ہو سکا۔
اب میں اپنی بات کو یہاں سمیٹوں گا کہ آپ ان تمام باتوں کا جواب تلاش کریں اگر آپ کے پاس ان باتوں کا جواب ہے تو تب بھی آپ یہاں ضرور اپنی معلومات شیئر کریں۔
والسلام
آپ کا دوست

 

پانچ وقت کی نماز قائم کرنے اور ہر وقت با وضو رہنے کی کوشش سے ایسا ہو سکتا ہے آزمائش شرط ہے

بے شک نماز بے حیائی اور برے کاموں سے روکتی ہے​

 

مصالحے دار اور چٹپٹ غذائیں خصوصًا لال مرچ کھانے سے احتراز کریں یہ جذبات میں برانگیختی پیدا کردیتی ہیں۔ نظروں کو کنٹرول کریں۔ نماز پڑھنے کے باوجود اگر آپ آج کل کی لوفر انڈین پاکستانی فلمیں بھی دیکھیں گے تو نماز بےبس ہوگی برائی اور بے حیائی روکنے میں۔

 

مناسب

آپ دونوں دوستوں نے حقیقت میں ساتھی اور دوست ہونے کا مطلب واضح کیا ہے۔ بہت شکریہ
اگر آپ میری پوری لڑی کو باغور پڑھتے تو آپ کا اظہار خیال کے لیے کچھ اور بھی موضوعات ملتے ۔میں ہماری اردو پیاری اردو کے باقی دوستوں سے گذارش کروں گا کہ وہ اس لڑی کو مکمل طور پر پڑھ کر اس میں موجود سوالات کے جواب دیں۔ جن کے لیے میں نے خاص طور پر یہ لڑی بنائی ہے۔

والسلام​

 

جس چیز سے آپ متاثر لگتے ہیں وہ یا تو یوگا کے اصول ہیں یا پھر کسی عامل ٹائپ بندے کے خیالات۔ ارتکاز پیدا کرنا، جنسی جذبہ کس طرح طاقت پیدا کرتا ہے انھیں شعبوں میں کام آتا ہے۔ یوگیوں کا کہنا ہے کہ اسے کنٹرول کرکے بندہ کنڈلینی شکتی کو بیدار کرلیتا ہے۔ اور عملیات کے دوران بھی جلالی اور جمالی پرہیز بتائے جاتے ہیں یعنی دوران عمل نا تو گوشت لہسن پیاز حیوانی چکنائی کھانی ہے نہ ہی بیوی کے قریب جانا ہے۔
یہ انھی لوگوں کی باتیں ہیں عام زندگی میں ان کا اطلاق کم ہی ہوتا ہے۔
جہاں تک آپ کا سوال کہ یہ کیسے پیدا ہوتا ہے تو یہ ہماری فطرت ہے۔ صحیح جواب کوئی ڈاکٹر صاحب ہی دے سکتے ہیں لیکن مجھے جہاں تک معلوم ہوا ہے کوئی پندرہ بیس دن کا ایک پراسس ہے جس میں آپ کے جسم میں (مردانہ) تولیدی مادہ تیار ہوتا ہے۔ اس کے بعد اگر اسے ایک خاص مدت (کتنی ہے کہہ نہیں سکتا) تک اس کا اخراج نہ ہو تو آپ کو قربت کی ضرورت محسوس ہوتی ہے۔ نہ ہونے کی صورت میں نارملی( قریبًا ایک ماہ میں ایک بار) رات احتلام ہوجاتا ہے۔ یہ آغاز شباب اور جوانی کے دور میں ہوتا ہے۔ وقت کے ساتھ ساتھ یہ بھی ماند پڑتا چلا جاتا ہے۔
یہ صرف ایک نارمل نوجوان کی کیفیات بتائی گئی ہیں جتنی میرے علم میں تھیں۔ جیسا کہ میرے پچھلے پیغام سے ظاہر ہے کہ خلاف فطرت اعمال کی صورت میں اس سے الگ معمول بھی بن سکتا ہے۔ کوئی ڈاکٹر یا حکیم صاحب ہوں تو اس سلسلے میں مزید بہتر رہنمائی کرسکتے ہیں۔ میں اگر کہیں غلط ہوں تو تصحیح فرما دیں۔ یہ معلومات نیک نیتی اور صرف اور صرف علم میں اضافے کے پیش نظر انتہائی خلوص نیت سے فراہم کی گئی ہیں۔
وسلام[/i]

 

امریکن بھائی نے لکھا ہے

حضور نبی اکرم صلی اللہ علیہ وسلم نے فرمایا “جو شخص تم میں سے عائلی ذمہ داری کا بوجھ اٹھانے کی ہمت رکھتا ہو اسے چاہیے کہ نکاح(شادی) کر لے کیونکہ یہ (عمل) نگاہ نیچی رکھنے والا اور شرمگاہ کی حفاظت کرنے والا ہے۔ اور جو شخص اس (گھربار سنبھالنے) کی طاقت نہ رکھتا ہے اسے چاہیے کہ روزہ رکھے کیونکہ روزہ اس کی شہوت کو کم کرنے والا ہوتا ہے۔“ (الحدیث)
میرے بھائی آپ نے لکھا کہ بحمد اللہ میں مسلمان ہوں ۔ اس میں کوئی شک نہیں کہ بحمد اللہ ہم سب مسلمان ہیں لیکن مسلمان کس درجہ کے؟ برائے نام؟ صرف اس لیے کہ مسلمان کے گھر پیدا ہو گئے؟ یا نام اسلامی ہے؟ یا اسلامی معاشرے میں رہتے ہوئے کسی اور سمت جانے کی ہمت نہیں؟
میرے بھائی اسلام کے لغوی مفہوم میں سے ایک معنیٰ اللہ کے حضور خود سپردگی Self-Submission to Allah ہے یعنی اسکے احکامات جو اس نے اپنے پیارے نبی اکرم صلی اللہ علیہ وسلم کے ذریعے سے ہم تک پہنچائے ہم ان کو سچ مان کر ان کو دل و جان سے تسلیم کر کے خود کو اللہ اور اسکے رسول صلی اللہ علیہ وسلم کے حوالے کر دیں اور اپنی زندگی کو انکے احکامات کے تحت اور تابع کر دیں تب ہم “حقیقی مسلمان“ کہلانے کے حقدار ہیں۔
مسلمان کی یہ تعریف سمجھ لینے کے بعد اب اوپر دی گئی حدیث کو ذرا پھر سے پڑھیں اور اپنے کہے گئے فرمودات کا موازنہ بھی کریں۔ یہ ایمان ہر مسلمان کا ہونا چاہیے کہ دنیا بھر کے سائنسدان ملکر بھی اس حقیقت کا ادراک شاید نہ کرسکیں جو اللہ تعالی کے محبوب نبی پاک صلی اللہ علیہ وسلم نے فرما دیا ہے۔ لیکن جو حقائق ہمارے محبوب نبی صلی اللہ علیہ وسلم نے بیان فرمائے اس کی تصدیق آج کی سائنس بھی حرف بحرف کرتی جا رہی ہے۔ آئیے ذرا مندرجہ ذیل لنک کا مطالعہ فرمائیں تاکہ عقل پسند مادی سوچ بھی اس حقیقت سے آگاہ ہو سکے۔

[

quote][font=Times New Roman, serif][align=left:1bgxn4zw][align=right:1bgxn4zw][align=left]Fasting Suppresses Pulsatile Luteinizing Hormone (LH)
Secretion and Enhances Orderliness of LH Release in
Young but Not Older Men*
MATTI BERGENDAHL, JOSEPH A. ALOI, ALI IRANMANESH,
THOMAS M. MULLIGAN, AND JOHANNES D. VELDHUIS
Departments of Pediatrics and Physiology (M.B.), University of Turku, FIN-20520 Turku, Finland; Division of
Endocrinology (J.A.A., J.D.V.), Department of Internal Medicine, National Science Foundation Center for
Biological Timing, University of Virginia Health Sciences Center, Charlottesville, Virginia 22908; Endocrine
Section Medical Service (A.I.), Salem Veterans Affairs Medical Center, Salem, Virginia 24513; and Geriatrics
Medicine (T.M.M.), Hunter Holmes McGuire Veterans Affairs Medical Center, Richmond, Virginia 23249
ABSTRACT
Pulsatile gonadotropin secretion and sex-steroid concentrations are
suppressed reversibly in young fasted or malnourished human subjects.
In this study, we investigated the impact of age on the dynamic neuroendocrine
mechanisms underlying this stress response in healthy
young (age, 28 6 3 yr, n 5 vs. older (age 67 6 2 yr, n 5 men with
similar body mass indices (mean, 26 6 0.6 vs. 26 6 1.3 kg/m2, respectively).
Serum LH concentrations were measured by immunoradiometric
assay (IRMA) in blood collected at 10-min intervals over 27 h on a
control (fed) day and on the third day of a 3.5-day fast (water only)
assigned in randomized order. After 24 h of basal sampling, GnRH (10
mg iv bolus) was administered to test gonadotrope responsiveness. Cortisol,
dehydroepiandrosterone sulfate, androstenedione, testosterone,
FSH, GH, and PRL were measured in 24-h pooled serum as positive and
negative control hormones. Approximate entropy was used to quantitate
the orderliness of LH release over 24 h, and a multiple-parameter deconvolution
method was applied to quantify pulsatile LH secretion and
LH half-life. In the fed state, older men exhibited elevated mean (24-h
pooled) serum FSH and cortisol concentrations compared with young
controls but equivalent serum LH concentrations and reduced serum
GH, free testosterone, androstenedione, and dehydroepiandrosterone
sulfate concentrations. Fed older men also manifested a lower frequency
and amplitude of 24-h pulsatile LH secretion, and, by approximate entropy
calculations, a more disorderly pattern of basal LH release than
younger individuals. Three- and one-half days of fasting evoked 40% and
47%increases inmean(24-h) serum cortisol concentrations in young and
older men, respectively (P , 0.01 vs. fed, but P 5 not significant for
percentage rise in older vs. young men). Concurrently, fasting induced
a 2.1-fold fall in the 24-h endogenous LH production rate in young men
(fed 36 6 9.7 vs. fasted 17 6 2.0 IU/L of distribution volume/day, P ,
0.01), but did not significantly affect the daily LH secretion rate in older
men(fed 2764.5 vs. fasted 2163.4 IU/day). The reducedLHproduction
rate in fasting young men was accounted for by a 1.7-fold decline in the
mass of LH secreted per burst (fed 2.5 6 0.45 vs. fasted 1.5 6 0.16 IU/L,
P , 0.05), whereas LH burst mass in older men remained unchanged
(and low) during fasting. In addition, in young men, during the 3.5-day
fast the number of computer-resolved LH secretory bursts per 24 h
decreased (fed 1560.7 vs. fasted 1160.7, P,0.01), and the interburst
interval increased (fed 94 6 4.2 vs. fasted 125 6 8.7 min, P , 0.05). In
contrast, in older men in the fed state, basal LH peak frequency and
serum free testosterone concentrations were reduced compared with
corresponding values in young men, and did not decline further with
fasting. Whereas the orderliness of LH release patterns increased significantly
during fasting in the young men, the approximate entropy
measure failed to change significantly in unfed older subjects. By cosinor
analysis, young men showed lower 24-h mesor (mean of nyctohemeral
rhythm of) serum LH concentrations than older volunteers during fasting.
Moreover, young but not older men manifested preserved 24-h variations
in LH interpulse intervals when fasting. Exogenously stimulated
LH release (mean 3-h serum LH concentration or calculated mass of LH
secreted) following a single iv injection of 10 mg GnRH was independent
of age and fasting status. We conclude that the metabolic stressor of
short-term fasting unmasks specific age-related neuroendocrine contrasts
in the stress-responsive control of both the pulsatile and nyctohemeral
regulation of the male hypothalamo-pituitary-gonadal-axis.
(J Clin Endocrinol Metab 83: 1967–1975, 199
Received November 21, 1997. Revision received February 12, 1998.
Accepted February 24, 1998.
Address all correspondence and requests for reprints to: Johannes D.
Veldhuis, Division of Endocrinology and Metabolism, Department of
Internal Medicine, Box 202 University of Virginia Health Sciences Center,
Charlottesville, Virginia 22908. E-mail: JDV@Virginia.Edu.
* This work was supported in part by NIH Grant RR-00847 to the
Clinical Research Center of the University of Virginia; NIH Reproduction
Research Center P30 HD 28934; Research Career Development
Award 1-KO4-HD-00634 (to J.D.V.); 1-FO5-TWO5080 from the Fogarty
International Center (FIC) of the NIH (to M.B.); Veterans Administration
(VA) Merit Review Medical Research Funds (to T.M.); the Baxter Healthcare
Corporation, Round Lake, IL (to J.D.V.); the Academy of Finland (to
M.B.); the Yrjo¨ Jahnsson Foundation (to M.B.); the Emil Aaltonen Foundation
(to M.B.); the University of Virginia Pratt Foundation and Academic
Enhancement Program (to J.D.V.); and the National Science Foundation
(NSF) Science Center in Biological Timing (to J.D.V.). The
contents of this publication are solely the responsibility of the authors
and do not necessarily represent the views of the VA, FIC, NIH, or NSF.
INADEQUATEnutritional intake for any particular level of
whole-body energy utilization alters neuroendocrine activity
in the healthy young human, e.g. by suppressing pulsatile
activity of the reproductive axis, and stimulating various
components of the stress-responsive somatotropic and
corticotropic axes (1–3). Thus, short-term nutritional deprivation
specifically decreases serum gonadotropin and gonadal
sex-steroid hormone concentrations, while amplifying
GH, ACTH, and cortisol secretion. Available mechanistic
studies in the young healthy human and experimental animals
suggest that reduced release of hypothalamic GnRH
plays a key role in the acute fasting-associated suppression
of reproductive function in both the male and female (1).
Clinical investigations in young men using repeated blood
sampling, LH immunoradiometric assay (IRMA), and deconvolution
analysis have shown that a 3.5-day fast elicits an
0021-972X/98/$03.00/0 Vol. 83, No. 6
Journal of Clinical Endocrinology and Metabolism Printed in U.S.A.
Copyright © 1998 by The Endocrine Society
1967
Downloaded from jcem.endojournals.org on October 18, 2006
approximately 50% decrease in mean (24-h) serum LH and
free testosterone concentrations, with an attendant reduction
in the daily LH secretion rate ascribable to two mechanisms:
first, a fall in the apparent number of computer-resolved LH
secretory bursts; and second, a decrease in the mass of LH
secreted per burst (4). Recently, it has been shown that pulsatile
iv GnRH infusions over the last 24 h of calorie withdrawal
can completely prevent the fasting-induced decline
in LH secretory burst mass and frequency (without altering
the increase in serum cortisol concentrations), and restore
serum total and free testosterone concentrations to baseline
(fed) values (4). The ability of pulsatile GnRH infusions alone
to overcome this form of stress-induced (in males) hypogonadism
supports the pathophysiological hypothesis that nutrient
withdrawal reversibly decreases output of the human
hypothalamic GnRH pulse generator. Thus, the metabolic
stressor of fasting in young men is a useful experimental
model for short-term hypothalamic GnRH deficiency mediating
hypogonadotropic hypogonadism.
Healthy aging in men also results in changes in the LH
pulse signal (5–13), as well as in the steroidogenic responsiveness
of the testis to gonadotropin challenges (14). Using
deconvolution analysis to calculate underlying gonadotropin
secretion rates and LH half-life, we observed that LH
secretory burst amplitude and mass decrease progressively
with increasing age (15). The clinical relative hypogonadism
of aging is accompanied by a decrease in serum total and free
testosterone concentrations (16 –20). Some of these features of
the aging GnRH-LH-testosterone axis are similar qualitatively
to those of stress responses of the young male reproductive
axis (above). However, whether age-related changes
in the hypothalamic-pituitary-gonadal axis reflect undue
susceptibility of older individuals to stress-associated inhibition
of the reproductive axis is not known. Moreover, to our
knowledge, there are no data available to define the relative
impact of a metabolic stressor, such as acute nutrient withdrawal,
on gonadotropin secretion in older vs. young men
(14).
In the present study, we investigated the effects of shortterm
fasting on the hypothalamic-pituitary-Leydig cell axis
in eight healthy young and eight older men. The metabolic
stressor of acute fasting was used as an experimental paradigm
to investigate the hypothesis that age alters the ability
of the dynamic (pulsatile and nyctohemerally rhythmic) reproductive
axis to respond to an inhibitory stressor. Selected
other hormones were measured in 24-h serum pools as positive
and negative controls.
Subjects and Methods
Clinical protocol
Eight young and eight older healthy men [body mass indices (BMIs)
2660.9 kg/m2, age 2863 yr, range 22–44 yr in the group of young men;
and BMI 26 6 1.3 kg/m2, age 67 6 2 yr, range 55–73 yr in older men]
were studied after provision of written informed consent approved by
the Human Investigation Committee of the University of Virginia. No
volunteer was taking medications, had undertaken transmeridian travel
for at least 1 week, or had recent weight loss. Each had a negative
detailed clinical history and physical examination with normal adult
sexual maturation and testicular size, normal screening biochemical
tests of renal, hepatic, metabolic, and hematological function, and unremarkable
(age-adjusted) morning serum concentrations of total and
free T4, TSH, GH, PRL, estradiol, free and total testosterone, immunoreactive
LH and FSH, and insulin growth factor-I (IGF-1). The pulsatile
LH data in six of the eight young controls were reported earlier (4). BMI
(but not visceral adiposity) was matched in the two study groups.
The volunteers were admitted to the General Clinical Research Center
of the University of Virginia the night before blood sampling both in the
fed state and again before 3.5 days of fasting. The fed and fasting
admissions were assigned in randomized order at least 1 month apart.
In both the fed and fasting studies, blood sampling was carried out at
10-min intervals for 27 h beginning at 0800 h at least 1 h after venipuncture.
Daytime naps were disallowed. In the 3.5-day fast, the 27-h
blood sampling interval occupied hours 56–83 (hour zero defined as
2400 h on the evening of admission). After blood sampling for 24 h, a
single bolus of 10 mg GnRH was given iv to test pituitary responsiveness
as assessed by 3 more h of 10-min blood withdrawal.
Blood samples were allowed to clot at room temperature, centrifuged,
and the subsequent sera frozen at2C for later assays. Subjects remained
in a bed or chair during sampling except for ambulation to the lavatory
as needed. In the fed state, three isocaloric meals were given per day (at
0800, 1200, and 1730 h). During the 3.5-day fast, the volunteers received
caffeine- and calorie-free liquids only, slept in the Clinical Research
Center, and had urinary ketones monitored twice to four times daily to
assess compliance with the fast. All patients had consistently positive
urinary ketones throughout the fast. Potassium chloride (40 mmol) and
water-soluble vitamins were administered orally daily, as described
earlier in other studies (2, 3, 21, 22)
Assays
Serum LH concentrations were measured robotically in each sample
in duplicate by a two-site IRMA (Nichols Labs., San Juan Capistrano,
CA), as described previously (15). This assay correlates well (P , 0.001)
with an in vitro Leydig cell LH bioassay over the range of LH concentrations
2–50 IU/L. The median inter- and intraassay coefficients of
variation were less than 8.5% for these studies. All 181 samples in each
admission were assayed together. The sensitivity of the assay was 0.20
IU/L, using the First International Reference Preparation. Serum total
and free testosterone, FSH, estradiol, dehydroepiandrosterone-sulfate
(DHEA-S), androstenedione, PRL, GH, IGF-1, IGF binding protein-3
(IGFBP-3), and TSH were assayed by RIA or chemiluminescent or immunoradiometric
assays, in a single 24-h pool of serum (50 mL aliquoted
from each of 145 samples) (2, 3, 15, 21, 23, 24).
Deconvolution analysis
Deconvolution analysis is a mathematical technique applied to a
pulsatile serum hormone concentration vs. time series to estimate subject-
specific measures of pulsatile hormone secretion and half-life (25–
27). The dailyLHsecretion rate was computed assuming negligible basal
LH secretion as the product of secretory burst frequency and the mean
mass of LH released per secretory pulse. Based on recent validation
studies in men, deconvolution analysis was carried out at 95% joint
statistical confidence intervals for all calculated LH secretory burst amplitudes
with the technician blinded to the randomized order of the fed
vs. fasted admissions. After deconvolving the entire 27-h time series of
serum LH concentrations, statistical analysis was applied separately to
the 24-h baseline (spontaneous pulsatile LH release) and the 3-h post-
GnRH (stimulated) segments.
Nyctohemeral (24-h) rhythmicity
Diurnal rhythms of serum LH concentrations as well as computed LH
secretory burst characteristics (mass per burst and interpulse interval)
were appraised using cosinor analysis, as described previously (21).
Approximate entropy (ApEn)
ApEn is a statistic that quantitates relative orderliness or regularity
of hormone release profiles. It complements usual pulse analysis, but
gives information about (sub)pattern recurrence or repetition within the
data. Pattern reproducibility is lost in tumoral hormone secretion and
reduced in aging. Loss of feedback control also is expected to elicit more
disorderly or irregular release patterns.
1968 BERGENDAHL ET AL. JCE & M² 1998
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ApEn comprises a family of model- and concentration-independent
statistics for assessing the apparent process randomness or serial irregularity
of a time series by quantifying the subpattern reproducibility not
necessarily identified by pulse-detection algorithms (2. A particular
ApEn statistic is a single finite nonnegative real number assigned as an
ensemble measure to a series of hormone concentrations with larger
ApEn values corresponding to relatively greater pattern randomness.
Specifically, ApEn measures serial data regularity or, technically, the
logarithmic likelihood that runs of patterns (of length m) that are similar
(within r) remain similar on next incremental comparison. The formal
definition of ApEn is given elsewhere (2. Two principal iut parameters,
namely m and r, are fixed to compute ApEn from vector sequences
constructed from the observed hormone concentration profiles, where,
m represents the window length of consecutive pattern measurements,
and r the tolerance or threshold for testing subpattern regularity. To
maintain scale invariance, r is typically fixed as a percentage of the total
(between-sample) sd of each hormone time series, e.g. 20%, and m as a
value of 1 or 2 denoting consecutive vectors of length 1 or length 2 data
points. In the present study, given 145 measurements of LH in each 24-h
time series, we calculated ApEn values with r 5 0.2 and m 5 1, which
provides the more appropriate statistic for assessing subpattern reproducibility
in data series of this size.
Statistical analyses
Differences between fed and fasted measures in young and older men
were assessed after logarithmic transformation using ANOVA followed
by Duncan’s multiple range test. Results are presented as the mean 6
sem. Statistical significance was accepted for a P value , 0.05 or for
nonoverlapping group 95% statistical confidence intervals (cosinor
analysis).
Results
Mean serum hormone concentrations
The 3.5-day fast in young men (n 5 resulted in a more
than 2-fold decrease in the mean (24-h) serum LH concentration
(IU/L, averaged over the 145 samples collected).
Older men showed no significant suppression (Table 1). In
particular, the mean (6 sem) fed vs. fasting serum LH concentrations
were: in young men, 3.5 6 1.0 vs. 1.6 6 0.16 IU/L
[fasting value P , 0.05 vs. young fed, and P 5 not significant
(NS) vs. older fed]; and, in older men, 4.260.69 vs. 3.160.47
IU/L (fasting value P , 0.01 vs. young fasting). Other measurements
were made in 24-h serum pools. Pooled serum
concentrations of total and free testosterone decreased, respectively,
by 40% and 46% in fasting young men (P , 0.01)
(Table 1). The corresponding androgen values were not
changed by fasting in older men, despite similar statistical
variances. Baseline (24-h pooled) serum free testosterone
concentrations were lower, and pooled serum FSH and cortisol
concentrations higher, in fed older vs. young men (Table
1). Serum pooled concentrations of cortisol increased significantly
and similarly during fasting in young and older men
(specifically by 40% and 47%, respectively, P , 0.01 vs. fed,
Table 1 and P 5 NS young vs. older for the percentage rises).
Serum pooled estradiol and IGFBP-3 concentrations were
statistically independent of age, whereas GH and DHEA-S
levels were lower (both P,0.01) at baseline (fed) in the older
cohort (Table 1).
Deconvolution analysis of pulsatile LH secretion
Illustrative 24-h profiles of serum LH concentrations,
which were pulsatile in all subjects in both the fed and fasting
studies, and deconvolution-resolved LH secretory rates from
one young and one older male are depicted in Fig. 1. The
specific quantitative changes induced by fasting in various
pulsatile attributes ofLHsecretion, as well asLHhalf-life, are
summarized in Table 2. Statistical analyses revealed that the
half-duration in minutes (the duration of the calculated secretory
event at half-maximal amplitude) of computed LH
secretory bursts decreased, but the calculatedLHhalf-life did
not change, significantly in response to fasting in young men.
In older men, the LH secretory burst half-duration and LH
half-life did not change during fasting, but both of these
parameters were greater at baseline and during fasting than
the corresponding values in young men. Individual half-life
data are given in Fig. 2A. The number of LH secretory pulses
per 24 h with statistically non-zero amplitudes (jointly at P,
0.05) fell significantly in fasting young men. In contrast, LH
secretory burst frequency was lower at baseline in fed older
men, and remained unchanged during fasting (Fig. 2B). Conversely,
the mean LH interpulse interval in young men rose
in response to fasting. In older men, the LH interburst interval
was not changed by fasting, and was greater in both
fed and fasted states than corresponding values in fed young
men. The mass of LH secreted per burst (area of the calculated
LH secretory pulse) in young men decreased significantly during
fasting, but this change was not evident in older men. There
were no significant alterations in computed LH secretory burst
amplitude (maximal rate of calculated LH secretion attained
within a release episode) in young or older men during fasting,
but the LH secretory pulse amplitude was lower in older men
in both dietary states compared with fed young men.
The product of the mass of LH secreted per burst and LH
TABLE 1. Mean (24-h) serum hormone concentrations (pools) in fed and fasting young and older men (n 5 8 in each group)
Hormone Young fed Young fasting Older fed Older fasting
FSH (IU/L) 2.4 6 0.46 1.8 6 0.18 6.1 6 1.40a 4.7 6 1.0b
Total testosterone (ng/dL) (to convert to nmol/L 3 0.0347) 581 6 70 343 6 42a 512 6 63 425 6 61
Free testosterone (pg/mL) (to convert to pmol/L 3 3.467) 23 6 4.7 13 6 1.0a 13 6 1.0a 11 6 1.2a
Estradiol (pg/mL) (to convert to pmol/L 3 3.671) 32 6 6.0 26 6 4.5 34 6 3.3 30 6 5.3
Androstenedione (ng/mL) (to convert to nmol/L 3 3.49) 1.1 6 0.04 1.1 6 0.06 0.8 6 0.12b 1.0 6 0.17
Cortisol (mg/dL) (to convert to nmol/L 3 27.6) 8.9 6 0.71 12.5 6 1.10a 11.4 6 0.59b 16.8 6 1.04c
DHEAS (mg/mL) (to convert to mmol/L 3 2.714) 484 6 63 654 6 39 98 6 17a 130 6 31a
GH (mg/L) 1.7 6 1.19 3.8 6 0.76a 0.4 6 0.10a 2.3 6 0.38d
IGF-1 (mg/L) 242 6 38 221 6 45 131 6 22 97 6 17b
IGFBP-3 (mg/L) 3249 6 273 3258 6 287 2778 6 127 2640 6 128
LH was determined in each of 169 samples in each volunteer, with 24-h means calculated from first 145 samples (pre-GnRH injections).
Data are mean 6 SEM, n 5 8 (ANOVA followed by Duncan’s multiple range test after logarithmic transformation).
a P , 0.01; b P , 0.05, compared with young fed controls; c P , 0.01; d P , 0.05, compared with older fed controls.
FASTING AND LH IN YOUNGER VS. OLDER MEN 1969
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secretory event frequency is the calculated 24-h pulsatile LH
secretion rate. In young but not older men, this measure
decreased in the fasted state.
Calculated LH secretory burst mass and the mean (3-h)
serum LH concentration following a single iv bolus injection
of 10 mg GnRH were both independent of fasting or age
(Table 3).
Nyctohemeral rhythmicity (cosinor analysis)
As shown in Fig. 3, A and B, young but not older men
exhibited significant 24-h variations in individual LH secretory
pulses mass in the fed state. Fasting reduced the mesor
(cosine mean) and amplitude of this rhythmicity in young
men but evoked a detectable rhythm in older men. Both
FIG. 1. Illustrative 24-h serum LH concentration (IRMA) profiles and calculated LH secretory rates as determined by deconvolution analysis
in one healthy young man and one older man studied in fed and fasted states. Blood samples were collected at 10-min intervals for 27 h when
volunteers were nutritionally replete (fed) and again during last 27 h of a 3.5-day fast. Fed and fasted sessions were assigned in randomized
order at least 4 weeks apart. GnRH (10 mg iv) was administered after first 24 h of baseline sampling. Baseline is shown over time 0–1440 min
(A and B), and post-GNRH responses over time 1440–1640 min (C and D). Continuous curves through observed serum LH concentrations (A
and C) are predicted by deconvolution analysis (see Subjects and Methods). Vertical bars through sample LH concentration values denote
dose-dependent intrasample SDS (estimated from all 191 replicated samples in each original 27-h time series). Punctuated episodes of
spontaneous LH release over 24 h or following GnRH iv (B and D) are computer-estimated LH secretory bursts (P , 0.05 vs. random sample
variance), which give rise to pulsatile serum LH concentration profiles. Calculated mean number, duration, mass, and amplitude of LH secretory
bursts and half-life of endogenous LH in eight young and eight older subjects each studied in both fed and fasted states are summarized in
Table 2. Table 3 gives LH responses to iv GnRH (10 mg).
TABLE 2. Deconvolution analysis of (24-h) pulsatile LH secretion and half-life in fed and fasting states in healthy young and older men
(n 5 8 in each group)
Parameter Young fed Young fasted Older fed Older fasted
LH secretory burst (half-duration (min) 7.8 6 1.53 4.4 6 1.05b 12.4 6 1.80b 10.1 6 1.44b
LH half-life (min) 84 6 2.8 91 6 6.0 149 6 9.7a,d 146 6 8.8a,d
LH secretory burst frequency (events/24 h) 15 6 0.71 11 6 0.73a 12 6 0.42a 12 6 0.75a
Interburst interval (min) 94 6 4.2 125 6 8.7b 119 6 3.9a 118 6 9.2a
LH secretory burst mass (IU/L) 2.5 6 0.45 1.5 6 0.16b 2.3 6 0.31 1.81 6 0.23
LH secretory burst amplitude (IU/L/min) 0.67 6 0.23 0.41 6 0.07 0.21 6 0.05b 0.22 6 0.06b
LH production rate/24 h (IU/L distribution volume/day) 36 6 9.7 17 6 2.0b 27 6 4.5 21 6 3.4
Data are mean 6 SEM, n 5 8 (ANOVA followed by Duncan’s multiple range test after logarithmic transformation).
a P , 0.01; b P , 0.05, compared with young fed controls; c P , 0.05, compared with older fed controls; d P , 0.01; e P , 0.05, compared with
young fasted.
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young and older men exhibited a significant 24-h variation
in LH interpulse interval in the fed state, which was abolished
in older men by fasting (Table 4).
As summarized in Table 5, cosinor analysis of serum LH
concentrations per se (rather than the above deconvolutioncalculated
parameters) revealed significant age-related contrasts
only in the fasting state, in which the mesor (mean)
reached a significantly lower value in young vs. older
volunteers.
ApEn
ApEn averaged 1.21 6 0.055 in fed young males, and fell
to 0.82360.089 during fasting (P,0.01) (Fig. 4). This change
indicates significantly greater orderliness or regularity of LH
release (lower ApEn value) in the fasting environment. Baseline
ApEn tended to be higher in older men at 1.43 6 0.054,
denoting greater irregularity of LH release over 24 h, as
observed earlier in overnight blood sampling (6). In contrast
to young men, older men’s ApEn values remained statistically
unchanged during fasting (1.16 6 0.074).
Discussion
The present investigation refutes our a priori hypothesis of
greater vulnerability of older men to the gonadotropin-suppressing
effects of short-term fasting. Indeed, fasting reduced
the secretion rate of LH and the serum free testosterone
concentration less in healthy older men. Moreover,
fasting enhanced the orderliness of LH release patterns (as
defined by the ApEn statistic) in young but not older men.
In contrast, young and older men had comparable adrenalgland
activation during fasting with, respectively, 40% and
47% (young and older) rises in mean (24-h) serum cortisol
concentrations. This suggests a similar adrenal stress response
(at least fractionally in young and older individuals).
As a positive control, both age groups showed severalfold
increases in (24-h pooled) serum GH concentrations in response
to fasting. Thus, the age distinction in neuroendocrine
adaptations to fasting was quite specific to the hypothalamopituitary-
gonadal axis.
Using deconvolution analysis, we reported earlier that a
3.5-day fast in young men brings about an approximately
50% fall in the calculated 24-h LH secretion rate. This was
neuroendocrinologically caused by a decline in both the apparent
number of computer-resolved LH secretory bursts
and the mass of LH secreted per burst (4). The suppressive
effect of fasting was completely reversed by pulsatile iv infusions
of GnRH. In the present study, we now observe that
in healthy older men short-term fasting fails to alter significantly
the 24-h LH production rate, LH pulse frequency, or
the mass of LH secreted per burst. Under stringent deconvolution-
fitting conditions of 95% joint confidence intervals,
baseline LH secretory burst frequency and amplitude (mass)
were both reduced at baseline in older men [the latter confirming
an evaluation earlier by 2.5-min blood sampling
overnight (29)], and remained low during fasting. The lower
(baseline) serum free testosterone concentration in older men
would tend to drive LH release (assuming normal negative
feedback), which might have opposed the tendency of fasting
to suppress the (older) axis.
Older men also were distinguished by more irregular or
disorderly patterns of 24-h LH release at baseline, reflected
in higher ApEn values [as also described independently in
6–8 h of overnight blood sampling (6)]. Moreover, in older
men, fasting failed to elicit significantly more orderly (lower
ApEn) LH release profiles, unlike the LH-gonadal-axis responses
identified in young men under the same study
conditions.
The capacity of gonadotroph cells to augment secretion of
biologically active LH during blockade of estrogen negative
feedback is decreased in older men (30, 31). On the other
hand, the secretion of bioactive LH in older men can be
amplified by treatment with a nonsteroidal androgen-receptor
antagonist (24). The latter finding indicates significantly
preservedGnRH/LHsecretory capacity in older individuals.
Taken together with other data, such observations allow for
the possibility (but do not prove) that aging is marked by
alterations in sex-hormone feedback control of the GnRHLH-
testosterone axis (32–35). In addition, we now describe
an apparent resistance to fasting-induced suppression of
both pulsatileLHsecretion and circulating (free) testosterone
concentrations in older men. This is consistent with an ageassociated
difference in adaptation of the GnRH-LH-Leydig
cell axis to a short-term metabolic stress. Whether this putative
resistance to fasting stress is related to the relative
FIG. 2. Individual LH half-life (A) and LH secretory burst frequency
(B) values in eight young and eight older men studied in fed vs. fasting
state for 3.5 days. Blood was sampled at 10-min intervals for 24 h and
assayed for LH content by IRMA. Deconvolution analysis was applied
to quantitate various LH secretory measures (Table 2) and half-life.
Numerical values are mean 6 SEM.
FASTING AND LH IN YOUNGER VS. OLDER MEN 1971
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inability of older men to increase pulsatile LH secretion during
treatment with an opiate-receptor antagonist (specifically
used to block opiate-dependent and hence putatively stressmediated
inhibition of GnRH secretion) (36) is not known. In
the rat, sex steroids significantly modulate the inhibitory
actions of both endogenous opiates and the stress of food
restriction on pulsatile LH release (37–39). The roles (if any)
of other putative hypothalamo-pituitary stress modulators
defined in the rat, such as CRH, neuropeptide Y, leptin, etc.
(1, 40, 41), in fasting-induced hypogonadotropism in men are
unestablished. In addition, whether and how estradiol, testosterone,
and/or endogenous opiate pathways modulate
fasting’s suppressive effects on GnRH secretion and/or its
feedback control in the young or older human are not known.
Because the type, duration, intensity, and novelty of various
stressors may influence the magnitude and nature of the
subsequent stress-adaptive responses, the present findings
of age-related contrasts in GnRH/LH secretory adaptations
to a particular metabolic-stress paradigm may or may not be
applicable in other stress contexts.
Fasting did not affect the calculated half-life of endogenous
LH in either group of men, but older men had an
apparently longer LH half-life at baseline and during fasting.
A longer half-life in older men could be because of reduced
LH removal, a larger LH distribution volume, altered LH
isoforms [e.g. more acidic (14) with reduced metabolic clearance],
and/or, on a technical basis, (unrecognized) basal LH
secretion or a greatly skewed LH waveform (42)]. We know
of no direct experimental data to distinguish among these
possibilities. Even so, fasting did not change the apparent
half-life of LH.
As reported previously using a simplified model of purely
pulsatile LH release (15), we found a significant prolongation
of the computed LH secretory-burst duration in older men.
LH secretory-burst duration also is altered in end-stage renal
failure in men (23), and increases in estrogen-treated postmenopausal
women (43). The intrapituitary and/or extraglandular
mechanisms underlying a prolonged (LH) secretory
event duration are not known. In this study, renal
function and serum estradiol concentrations were normal
and similar in both age groups (Table 1). On the other hand,
the significantly lower mean serum free testosterone concentration
in the older men could explain in part their calculated
longer LH half-life and/or LH secretory burst duration,
because short-term androgen deprivation with
flutamide will induce both of these changes (44).
Older men had more disorderly LH release than young
individuals, as quantified by ApEn. This was reported previously
based on overnight blood sampling every 2.5 min in
another group of older (vs. young) men (6). In this study, via
10-min sampling over 24-h, we found that ApEn values fall
in young men during fasting, signifying more orderly or
regular LH release at this time. In contrast, older men retained
more disorderly (24-h) LH release. On mathematical
grounds, the orderliness of hormone release is believed to
reflect the strength and/or complexity of key feedback interactions
within a neuroendocrine axis (6, 45). Thus, our
observations point to reduced feedback organization within
the older (male) hypothalamo-pituitary-Leydig cell axis at
baseline [present data and (6)], which fails to normalize with
fasting.
The suppression of LH secretory burst mass (and its 24-h
rhythmicity, Fig. 3A) in fasting young but not older men was
not attributable to measurable differences in pituitary gonadotrope-
cell responsiveness to GnRH injections in the two
age groups. Indeed, 10 mg GnRH iv stimulated similar LH
release independently of age or fasting. Recent iv GnRH
dose-LH secretory response analyses in (fed) young and
older men revealed enhanced maximal stimulatory effects of
GnRH (greater stimulus efficacy) in older subjects, with similar
half-maximal GnRH actions (similar agonist potency of,
or pituitary sensitivity to, GnRH) across age (46). Because
GnRH dose-LH response curves are not available in fasted
young and older individuals, we cannot exclude unequal
sensitivity to GnRH in fasting aged vs. young volunteers.
Masking of results by greater visceral fat [possibly suppressive
of LH pulse amplitude (15)] in older men also cannot be
excluded, because volunteers were matched for BMI only.
The older men in this study exhibited lower basal (fed)
serum GH, androstenedione, and DHEA-S concentrations,
and higher serum FSH and cortisol concentrations in 24-h
serum pools as expected, indicating representativeness of
these cohorts (14). In both older and young men, mean (24-h)
serumGHand cortisol concentrations rose significantly with
fasting corroborating prior data in young men and documenting
compliance with the fast (2, 3).
Nyctohemeral (24-h cosinor) rhythms in serum LH concentrations
showed reduced mesor (mean) values in fasted
young compared with older men, indicating suppressed
overall 24-h LH release. Separate cosinor analyses of (deconvolution-
computed) LH secretory burst mass disclosed
loss of 24-h rhythmic variation in young fasted (but not older
fasted) men, thus unmasking another age contrast. Both age
groups exhibited day-night variations in (deconvolution-calculated)
LH interburst intervals in the fed state, which were
abolished in older (but not young) fasted individuals. Although
the exact (presumptively neural) mechanisms that
generate such 24-h rhythmicities within the human LH axis
are not yet known (14), in this study we document age
differences both basally and fasting. Our LH data comple-
TABLE 3. Deconvolution analysis of GnRH-stimulated LH secretion over 3 h immediately following a single iv bolus injection of 10 mg
GnRH in fed and fasting states in healthy young and older men
LH measure Young fed Young fasted Older fed Older fasted
LH secretory burst mass (IU/L/burst) 6.5 6 0.73 15 6 4.1 9.6 6 2.1 13 6 2.6
3-h mean serum LH concentration (IU/L) 6.0 6 0.53 8.7 6 1.9 9.1 6 1.5 10 6 2.0
3-h integrated LH level (IU/L 3 min) 1140 6 98 1650 6 360 1660 6 282 1870 6 360
GnRH-stimulated LH release significantly above preinjection baseline (or 24-h mean) in all groups studied.
Data are mean6SEM, n58/group (ANOVA followed by Duncan’s multiple range test after logarithmic transformation revealed no significant
effects of age or dietary state on the measures shown).
1972 BERGENDAHL ET AL. JCE & M² 1998
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FIG. 3. Nyctohemeral (24-h) rhythmicity of deconvolution-calculatedLHsecretory burst mass (A) andLHinterpulse intervals (B) for all subjects
combined in each of two groups, specifically young (n 5 vs. older (n 5 men, as assessed by cosinor analysis of deconvolution measures
(Subjects and Methods). NS, Not significant (P , 0.05) amplitude of 24-h periodic fit. Fitted amplitudes (and 95% statistically confidence
intervals) are noted for significant group rhythms.
FASTING AND LH IN YOUNGER VS. OLDER MEN 1973
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ment an earlier report of diminished testosterone rhythmicity
over 24 h in (fed) older men (47).
In summary, fasting in young but not older men reduces
24-h LH secretory burst frequency and mass and (24-h
pooled) serum free testosterone concentrations. Fasting in
young but not older men enhances the quantifiable orderliness
(lower ApEn) of LH release, and alters 24-h LH rhythmicity.
Thus, we conclude that the metabolic stress of shortterm
fasting unmasks age-related dynamic differences in the
pulsatile, 24-h rhythmic, and orderly release of LH.
Acknowledgments
We thank Patsy Craig for her skillful preparation of the manuscript;
Paula Azimi for her data analysis, statistical assistance, and artwork;
Brenda Grisso for performance of the immunoassays; and Sandra Jackson
and the expert nursing staff at the University of Virginia General
Clinical Research Center for conduct of the research protocols.
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15. Veldhuis JD, Urban RJ, Lizarralde G, Johnson ML, Iranmanesh A. 1992
Attenuation of luteinizing hormone secretory burst amplitude is a proximate
TABLE 4. Cosinor analysis of deconvolution measures in fed vs. fasting young versus older men
Amplitude Phasea Mesorb
LH secretory burst mass (IU/L)
Fed young 0.50 (0.02– 0.9c 34 (2186–261)c 2.6 (2.3–2.9)c
Fed older NS NS 2.3 (2.0 –2.6)c
Fasting young NS NS 1.6 (1.4 –1.9)c
Fasting older 0.49 (0.057–0.93)c 136 (62–340)c 1.9 (1.6 –2.2)c
LH interburst interval (min)
Fed young 12 (3.2–21)c 292 (110–474)c 94 (87–101)c
Fed older 11 (0.77–22)c 352 (101–620)c 119 (111–127)c
Fasting young 21 (1.3–40)c 644 (437–836)c 114 (102–127)c
Fasting older NS NS 115 (103–126)c
a Acrophae, Time in min (before 0800) of the maximum of 24-h rhythm.
b Mesor or mean of 24-h rhythm.
c 95% group confidence interval for each parameter.
NS, Significant amplitude, and hence no determinable phase.
TABLE 5. Serum 24-h LH concentration profiles (not fitted
parameters of secretion per se): cosine fits of 24-h rhythmicity
Group Amplitude Phase Mesor
Young fed 0.77 6 0.350 740 6 172 2.9 6 0.96
Older fed 0.49 6 0.056 1030 6 141 3.9 6 0.66
P NS* NS NS
Young fasting 0.43 6 0.048 780 6 160 1.0 6 0.16
Older fasting 0.37 6 0.054 510 6 140 2.6 6 0.41
P NS NS P , 0.01
Means 6 SEM of individual fits, n 5 8 young, and n 5 9 older men
(Wilcoxon uaired two-tailed test: NS, denotes P $ 0.05.)
a One-half the difference between zenith and nadir values.
b Time in minutes (before 0800 h) of maximum value in 24-h
rhythm.
FIG. 4. Individual ApEn values of 24-h serum LH concentration profiles
in fed vs. fasted young (n 5 vs. older (n 5 men. ApEn is a
relative measure of disorderliness or irregularity of hormone release,
with higher absolute values denoting greater irregularity. Conversely,
lower ApEn values as observed in this study in young fasted
men indicate more orderly patterns. Data are presented as indicated
in legend of Fig. 2.
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42. Veldhuis JD, Evans WS, Johnson ML. 1995 Complicating effects of highly
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44. Veldhuis JD, Zwart AD, Iranmanesh A. 1997 Neuroendocrine mechanisms by
which selective Leydig-cell castration unleashes increased pulsatile LH release
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45. Veldhuis JD, Metzger DL, Martha Jr PM, et al. 1997 Estrogen and testosterone,
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FASTING AND LH IN YOUNGER VS. OLDER MEN 1975​

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سو میرے بھائی اب اگر حضور نبی اکرم صلی اللہ علیہ وسلم کے فرمان کے مطابق ہم روزہ کے ذریعے جنسی جذبہ کنٹرول نہیں کر سکتے تو کم از کم سائنسی اصول کو ہی مانتے ہوئے یہ حقیقت تسلیم کرنا پڑے گی کہ روزہ سے جنسی جذبہ کم ضرور ہوتا ہے۔ بشرطیکہ روزہ رکھنے کے تمام آداب و شرائط پورے کیے جائیں۔ یہ ایک الگ موضوع ہے کہ روزہ کے ظاہری اور باطنی آداب و شرائط کیاہیں؟ جب تک کوئی عمل اپنے پورے تقاضوں کے ساتھ نہیں کیا جاتا اس کا نتیجہ بھی کما حقہُ نہیں نکل سکتا۔ ہمیں مان لینا چاہیے کہ اگر فرمان ِ رسول صلی اللہ علیہ وسلم اور سائنسی اصولوں کی وضاحتوں کے باوجود بھی ہم مطلوبہ نتائج نہیں پا سکتے تو یہ قصور ہمارا ہے نہ کہ مسلمانی کا۔ ہمیں چاہیے کہ تصوف کو مطالعہ کریں اسے سیکھیں ۔ ایمان مضبوط کریں قیامت اور آخرت کے کثرت سے یاد رکھیں۔ اسلام و ایمان کو قلب و جان میں اتار لیں۔ جب ہم اللہ اور اسکے رسول صلی اللہ علیہ وسلم کے قائم کردہ معیارِ مسلمانی پر پورا اتریں گے تو پھر ہمیں احکاماتِ اسلامی کے نتائج بھی ملنا شروع ہو جائیں گے اور ہر طرح کے شیطانی اور نفسانی مشکلات سے چھٹکارا بھی مل جائے گا۔

 

میں نعیم بھائی کے خیالات کی تائید کرتا ہوں واقعی روزہ صرف بھوکے اور پیاسے رہنے کا نام نہیں

 

امریکن بھائی !!
نعیم بھائی کے دلائل میں وزن ہے۔ ماننا پڑے گا کہ قصور ہمارا ہی ہے۔ اسلام یا اسلامی تعلیمات کا نہیں۔

 

کیا یہ موضوع یہاں بہت ضروری تھا ؟

 

نادیہ خان جی میں آپ سے اتفاق کرتا ہوں۔ اگر میں اس فورم کا منتظم ہوتا تو شاید اس موضوع کو بلاک کر دیتا۔ لیکن یہ موضوع کچھ ایسا گناہ بھی نہیں۔ ہمیں حسنِ ظن رکھتے ہوئے امریکن بھائی کی نیت پر شک نہیں کرنا چاہیئے ممکن ہے انہوں نےاپنی کسی مشکل پر قابو پانے کے لیے یا اپنے علم میں اضافہ کے لیے اس موضوع پر دوسروں کی رائے طلب کی ہو۔ اگر ہمارے پاس کچھ معلومات اس ضمن میں ہیں تو ہمیں چاہیئے کہ دیانتداری اور خلوص سے انکی حتی المقدور مدد کریں۔ ویسے بھی تبادلہء خیالات کرنے سے علم میں اضافہ ہوتا ہے۔
البتہ یہ موضوع خواتین کے لیے موزوں نہیں ہے

 

ٹھیک ہے آپ کریں خدمت خلق اللہ حافظ

 

سوال کا جواب

میرے پیارے پیارے بھائی بہت شکریہ کہ تم نے اپنے خیالات سے نوازہ.

But

ابھی تک سب لوگوں نے ایک ہی سوال کا جواب دینے میں‌لگے ہوئے ہیں۔ آپ لوگ باقی سوالات کو بھی مدنظر رکھیں۔​

 

میں اپنے سوالات دوبارہ دہرا رہا ہ

میں اپنے سوالات کو دوبارہ دہرا رہا ہوں۔ برائے مہربانی ان سوالات پر غور کرنے کے بعد ان کا جواب دیجئے گا۔

اس طاقت یا جذبہ کے ذریعے جسمانی طاقت پیدا ہوتی ہے۔یہ جنسی طاقت کیسے پیدا ہوتی ہے؟
یہ کیسے ایک بندے کے اندر ذہنی ارتکاز۔ یکسوئی پیدا کرتی ہے۔اس جذبہ کے ذریعے ذہنی ارتکاز کیسےممکن ہوتا ہے؟
یہ کیسے ممکن ہے کہ ایک بندہ اپنے جنسی جذبہ کو کم از کم دو ماہ تک کنٹرول کر سکے؟ ​

 

آپ کے ذہن میں صرف ایک پریشانی کیوں سما گئی ہے؟؟؟
آپ کو اچھی طرح معلوم ہے کہ اس فورم میں بحمداللہ تعالیٰ سب مسلمان ہیں ۔اورہم روشن خیال ہونے کے باوجود کچھ الفاظ، کچھ اصطلاحات، اور کچھ موضوعات اخلاقی حدود و قیود کی وجہ سے اس طرح سرٍ عام زیرٍ بحث نہیں لا سکتے اسکے علاوہ یہ بات بھی نہیں بھولنی چاہییے کہ یہ فورم خواتین کے لیے بھی ہے ۔
امریکن بھائی ! براہ کرم ناراض نہ ہونا لیکن مجھے بتائیں کہ کیا دنیا بھر کی میڈیکل سائنس اور ریسرچ صرف جنسی جذبہ کے گرد ہی گھومتی ہے؟ کیا دنیا بھر میں اسکے علاوہ اور کوئی مسئلہ نہیں رہا؟ یا آپ کی زندگی میں اسکے علاوہ اور کوئی مسئلہ نہیں ہے؟
ہم سب آخر تھوڑی سی فکرباقی زندگی کی کیوں نہیں کر سکتے؟ہم کیوں نہیں پوچھ سکتے ہیں کہ :
شادی کے لیے بہترین عمر کیا ہے؟
انسان کو شادی کب کر لینی چاہیے؟
شادی کے بعد میاں بیوی کے حقوق فرائض ، اسلام میں کیا ہیں؟ بحثیت مسلمان ہمیں کیسے زندگی گذارنی چاہییے؟
معاف کیجیے گا آپ کے سوالات میں سوالات کم اور چسکہ بازی زیادہ ہے۔ آپ کو صرف جنسی جذبہ پیدا ہونےکے عوامل میں ہی تجسس کیوں ہے؟
انسان کے اندر بے شمار دوسرے جذبات بھی تو ہیں؟ آپ انکی فکر کیوں‌نہیں کرتے؟
یہ سوالات آپکے ذہن میں پیدا کیوں نہیں ہوتے کہ :
ہماری زندگی کا مقصد کیا ہے؟ جانور اور انسان کی زندگی میں کیا فرق ہے؟ اگر صرف سانس لینا، کھانا پینا، بچے پیدا کرنا ہی زندگی ہے تو جانوروں کے مقابلے میں انسان کو اشرف المخلوقات کیوں بنایا گیا؟
ہم مسلمان سچے دین پر ہونے کے باوجود دنیا میں ذلیل و خوار کیوں ہیں؟
مرنے کے بعد انسان کیسے حساب کتاب دے گا؟
کون کون سے اعمال قبر میں کام آ سکتے ہیں؟ قیامت کے دن کے حالات کیا ہوں گے؟ انسان کے اعضاء روزٍ محشر کیسے انسان کے اعمال کی گواہی دیں گے وغیرہ وغیرہ؟ کبھی یہ بھی سوچا یا سوچنے کی کوشش کی؟‌
‌امریکن بھائی !
آپ کو اس موضوع پر اتنی ہی معلومات درکار ہیں تو دنیا میں سینکڑوں کتابیں اس موضوع پر موجود ہیں۔ آپ آن لائن بھی درجنوں لنکس اور سائٹس کے ذریعے اپنی معلومات میں اضافہ کر سکتے ہیں۔ مثال کے طور پر http://sexuality.about.com/od/sexualhealthqanda یا اور اسطرح کے کئی لنک آپ کو Search Engine سے مل سکتے ہیں۔
میں امید کرتا ہوں کہ آپ میری باتوں پر ٹھنڈے دل سے غور کرنے کے بعد فراغ دلی کا مظاہرہ کرتے ہوئے خود بھی اور دیگر لوگوں کو بھی اس بحث میں الجھا کر وقت ضائع کرنے کی بجائے اس فورم کو مثبت طریقے سے استعمال کریں گے۔ شکریہ